Home | Current Issue | Past Issues | Search | CollectionsRSS | PDA Services | FAQ | SHCIM Online | Chinese Updated Wednesday, December 07, 2016
 Services
Submit a manuscript
Subscribe
Individuals
Institutions
Response
Send response
Read responses
Reprint
Advance online publication
Sign up for e-alert
JCIM job opportunities
Contact JCIM
 Information For
Readers
Authors
Reviewers
Advertisers
 About JCIM
Journal description
Editorial policy
Privacy policy
Copy right
Editorial staff
 Language Polishing
Journal of Chinese Integrative Medicine: 2008; 6(11): 1105-1108
DOI: 10.3736/jcim20081102
Combination and transformation of toxin and blood stasis in etiopatho-genesis of thrombotic cerebrocardiovascular diseases
1. Da-zhuo SHI (Cardiovascular Center, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing 100091, China E-mail: shidz666@sohu.com)
2. Hao XU (National Integrated Center of Cardiovascular Disease, China-Japan Friendship Hospital, Beijing 100029, China )
3. Hui-jun YIN (Cardiovascular Center, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing 100091, China )
4. Jing-chun ZHANG (Cardiovascular Center, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing 100091, China )
5. Ke-ji CHEN (Cardiovascular Center, Xiyuan Hospital, China Academy of Chinese Medical Sciences, Beijing 100091, China )
Abstract: According to the basic theory of traditional Chinese medicine (TCM), the pathogenetic factors such as platelet activation, adhesion, congregation and thrombosis fall into the category of blood stasis, while the pathological changes such as tissue necrosis, oxidative stress injury and inflammation, etc, are far beyond the etiological category of blood stasis. The toxin or the combination and transformation of toxin and blood stasis of TCM are involved in the pathogenesis of thrombotic cerebrocardiovascualr diseases. It is significant to recognize and stress the combination and transformation of toxin and stasis in pathogenicity so as to enrich TCM etiology and improve TCM clinical efficacy in the treatment of cerebrocardiovascular and thrombotic diseases.
Welcome to JCIM! You are the number 9305 reader of this article!
Download Article:
[Full Text]      [PDF]      [Chinese]      [Previous]      [Next]      [This Issue]
Please cite this article as:
Shi DZ, Xu H, Yin HJ, Zhang JC, Chen KJ. Combination and transformation of toxin and blood stasis in etiopatho-genesis of thrombotic cerebrocardiovascular diseases. J Chin Integr Med / Zhong Xi Yi Jie He Xue Bao. 2008; 6(11): 1105-1108.
References:
1Chen KJ, Li LD, Weng WL, et al. Studies of blood stasis syndrome and activating blood circulation[J].Zhongguo Zhong Xi Yi Jie He Xin Nao Xue Guan Bing Za Zhi, 2005, 3(3): 1-2. Chinese.
2Li PT, Wang YY, Huang QF. The hypothesis of the injury of brain collaterals by toxins and its theoretical and practical significance[J].Beijing Zhong Yi Yao Da Xue Xue Bao, 2001, 24(1): 1-6. Chinese with abstract in English.
3Chi MY. Integration of Chinese and Western medicine thrombology[M]. Beijing: People's Medical Publishing House, 2004. 1-5. Chinese.
4Wagner DD, Burger PC. Platelets in inflammation and thrombosis[J].Arterioscler Thromb Vasc Biol, 2003, 23(12): 2131-2137.  .
5Ruggeri ZM. Platelets in atherothrombosis[J].Nat Med, 2002, 8(11): 1227-1234.  .
6Pearson TA, Mensah GA, Alexander RW, et al. Markers of inflammation and cardiovascular disease: application to clinical and public health practice: A statement for healthcare professionals from the Centers for Disease Control and Prevention and the American Heart Association[J].Circulation, 2003, 107(3): 499-511.  .
7Kovanen PT, Kaartinen M, Paavonen T. Infiltrates of activated mast cells at the site of coronary atheromatous erosion or rupture in myocardial infarction[J].Circulation, 1995, 92(5): 1084-1088.  .
8Feinbloom D, Bauer KA. Assessment of hemostatic risk factors in predicting arterial thrombotic events[J].Arterioscler Thromb Vasc Biol, 2005, 25(10): 2043-2053.  .
9Li JZ. Attention to arterial thrombotic diseases[J].Zhonghua Nei Ke Za Zhi, 2006, 45(1): 2-3. Chinese.
10Porter TR, Li S, Oster R, et al. The clinical implications of no reflow demonstrated with intravenous perfluorocarbon containing microbubbles following restoration of Thrombolysis In Myocardial Infarction (TIMI) 3 flow in patients with acute myocardial infarction[J].Am J Cardiol, 1998, 82(10): 1173-1177.  .
11Kenner MD, Zajac EJ, Kondos GT, et al. Ability of the no-reflow phenomenon during an acute myocardial infarction to predict left ventricular dysfunction at one-month follow-up[J].Am J Cardiol, 1995, 76(12): 861-868.  .
12Wei JL. Hypothesis on the relationship between toxic heat and apoplexy[J].Beijing Zhong Yi Yao Da Xue Xue Bao, 2003, 26(1): 7-11. Chinese.
13Lu XH. Clinical effect and mechanism on treating unstable angina pectoris by Huanlian Jiedu Capsule[J].Shandong Zhong Yi Yao Da Xue Xue Bao, 2005, 29(6): 457-460. Chinese with abstract in English.
14Wen C, Xu H, Huang QF, et al. Effects of herbs of activation blood on atherosclerotic plaque morphology in ApoE gene-deficient mice[J].Zhongguo Bing Li Sheng Li Za Zhi, 2005, 21(8): 1640. Chinese.
15Wen C, Xu H, Huang QF, et al. Effect of drugs for promoting blood circulation on blood lipids and inflammatory reaction of atherosclerotic plaques in ApoE gene deficiency mice[J].Zhongguo Zhong Xi Yi Jie He Za Zhi, 2005, 25(4): 345-349. Chinese with abstract in English.

 Home | Current Issue | Past Issues | Search | CollectionsRSS | PDA Services | FAQ | SHCIM Online | Chinese
Copyright © 2003-2012 by JCIM Press. All rights reserved. ISSN 1672-1977